Skin Test for Alzheimer's, Parkinson's Shows Early Promise
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Humans' closest relative may also suffer from Alzheimer's
Humans are the only animal known to develop Alzheimers disease, and an official diagnosis requires checking off this list of three things: dementia, which is observed through screenings, and two pathologic markers—amyloid plaques (sticky bunches of misfolded proteins) and neurofibrillary tangles (tau proteins clumped together and twisted around).
L'effet protecteur de la caféine contre l'Alzheimer mieux compris
Brain disease affects 99% of NFL players in study
"The neurodegenerative brain disease CTE is typically found in people who have suffered repeated blows to the head, studies have found. It causes a build-up of so-called tau proteins which can disable neuro-pathways and cause memory loss, impaired judgment, confusion and a variety of other mental health issues."
Toxic Tau Of Alzheimer's May Offer A Path To Treatment
Hyperphosphorylation of the tau protein (tau inclusions, pTau) can result in the self-assembly of tangles of paired helical filaments and straight filaments, which are involved in the pathogenesis of Alzheimer's disease, frontotemporal dementia, and other tauopathies.
Antibody helps detect protein implicated in Alzheimer's, other diseases: May lead to novel ways to diagnose, monitor brain injury
Damaging tangles of the protein tau dot the brains of people with neurodegenerative diseases like Alzheimer's and boxer's dementia. Now, a team of scientists has found a way to measure tau levels in the blood that accurately reflects levels of tau in the brain. The study, in mice and a small group of people, could be the first step towards a non-invasive test for tau.
Measuring Neurofilament is a marker of nerve damage Bergman J Dring A Zetterberg H Blennow K Norgren N Gilthorpe J Bergenheim T Svenningsson A. Neurofilament light in CSF and serum is a sensitive marker for axonal white matter injury in MS. Neurol Neuroimmunol Neuroinflamm. 2016 Aug 2;3(5):e271. doi: 10.1212/NXI.0000000000000271. eCollection 2016 Oct. OBJECTIVE:In an ongoing open-label phase 1b study on the intrathecal administration of rituximab for progressive multiple sclerosis an…
Researchers at the Gladstone Institutes have shown that reducing brain levels of the protein tau effectively blocks the development of disease in a mouse model of Dravet syndrome, a severe intractable form of childhood epilepsy.
Tau normally acts as a molecular scaffold inside nerve cells, helping them to maintain order and structure and transport cargo along their long processes. In several forms of dementia, including Alzheimer’s, frontotemporal dementia, corticobasal degeneration and progressive supranuclear palsy, tau starts to behave out of character. Our blog explores the role of the protein tau and its role.
After examining more than 3,500 donated brains, scientists have gathered evidence to suggest that a rogue protein, tau, which has long been suspected to play a role in the development of Alzheimer’s, is the major driver of cognitive decline in this disease. These new findings downplay the role of amyloid, another dysfunctional protein that builds up as the condition progresses and has been the focus of much Alzheimer’s research.
Who takes care of their brain? I dare you to Google TAU PROTEIN and see what comes up. EHT helps optimize PP2A which helps regulate the TAU PROTEIN in the brain which is imperative to memory, mental clarity and OVERALL brain function. #SaveYourBrain #BrainHealth #SignumBioscience #Memory